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Sweet Syndrome in Children: A Clinician's Monograph

Clinician reference on Sweet syndrome (acute febrile neutrophilic dermatosis) — von den Driesch criteria, classical vs malignancy and drug-induced subtypes, red flags, and management.

Full criteria: Sweet Syndrome.

Definition and epidemiology

Sweet syndrome — acute febrile neutrophilic dermatosis — is a reactive inflammatory disorder defined by the abrupt onset of tender erythematous plaques, fever, and a dense dermal neutrophilic infiltrate without vasculitis. It is uncommon in childhood (roughly 5–8% of cases) and is conventionally split into three subtypes: classical/idiopathic (often post-infectious, especially upper-respiratory or gastrointestinal infection), malignancy-associated (in children chiefly haematological — leukaemia, myelodysplasia), and drug-induced (G-CSF, all-trans retinoic acid, certain antibiotics). A distinct neonatal/infantile presentation and association with immunodeficiency and autoinflammatory syndromes are recognised. Skewed female predominance seen in adults is less marked in children.

Clinical features

Onset is abrupt. Lesions are tender, sharply demarcated, juicy red-to-violaceous plaques, nodules, or pseudovesicles, typically on the face, neck, and upper limbs, sometimes with pustules, bullae, or a targetoid appearance; pathergy (lesions at sites of trauma) may occur. High fever and malaise are usual, and an elevated neutrophil count and acute-phase response are characteristic. Extracutaneous (neutrophilic) involvement — ocular (conjunctivitis, episcleritis), musculoskeletal (arthralgia, arthritis), pulmonary, hepatic, and CNS — can occur and is more concerning in children. Mucosal lesions, though rare, are described.

Diagnosis

Diagnosis rests on the von den Driesch (1994) criteria: both major criteria plus two of four minor criteria establish classical or malignancy-associated Sweet syndrome. The major criteria are (1) abrupt onset of tender/painful erythematous plaques or nodules and (2) a histology of dense dermal neutrophilic infiltrate without leucocytoclastic vasculitis — the histological cornerstone. The minor criteria are (1) fever > 38 °C; (2) association with an underlying haematological/visceral malignancy, inflammatory disease, pregnancy, or antecedent respiratory/GI infection or vaccination; (3) excellent response to systemic corticosteroids (or potassium iodide); and (4) abnormal labs at presentation — three of four of ESR > 20 mm/h, raised CRP, leucocytosis > 8,000, and neutrophils > 70%. Drug-induced Sweet syndrome uses a separate five-item set requiring a temporal drug relationship and resolution on withdrawal. Skin biopsy is essential to confirm the infiltrate and exclude infection and vasculitis. In every child, screen for an underlying trigger — including a haematological workup for occult malignancy.

See the full criteria: Sweet Syndrome Criteria

Red flags

Management overview

First, identify and treat the trigger — withdraw a culprit drug, treat the precipitating infection, and evaluate for malignancy. Systemic corticosteroids are the mainstay and produce a dramatic, often diagnostic, response with rapid defervescence and lesion resolution; a gradual taper limits relapse. For localised disease, potent topical or intralesional corticosteroids may suffice. Steroid-sparing or recurrent disease is managed with first-line alternatives such as potassium iodide, colchicine, or dapsone; refractory cases may need other immunomodulators or biologics. Doses are deliberately omitted here — verify weight-based regimens against current guidance. Most children respond well with a good prognosis, but malignancy-associated disease follows the course of the underlying condition.

References

Decision support for qualified clinicians only — verify against current primary guidelines and your clinical judgement.

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References

Last updated 2026-06-28.

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